PhD in Environmental Toxicology, University of
Wisconsin, Madison, 2001.
Additional specialization in
BA in Chemistry, Grinnell College, Grinnell, IA, 1996.
BA in German, Grinnell College, Grinnell, IA 1996
Department of Chemistry, Seattle University, Seattle, WA, 2003-present.
Research Fellow. Laboratory of Dr. Robin Wright. Department of Zoology,
University of Washington, Seattle, WA, October 2001 to August 2003.
Research Service Award Predoctoral Traineeship in Environmental Toxicology.
Environmental Toxicology Center, University of Wisconsin, Madison, WI, June 1997
to May 1999 and September 2000 to August 2001.
Department of Pathology, University of Wisconsin, Madison, WI, June 1999 to
The ERAD components Ubc7p,
Cue1p, and Doa10p are essential for viability in cells with increased
sterol biosynthetic capacity
Lynelle Larson, Mark Parrish, Aaron Sturm,
Christine Tachibana, Martin
Bard, Robin Wright (manuscript submitted).
A Low-Tech, High-Throughput Screen of the
Saccharomyces cerevisiae Deletion Mutant Collection Efficiently Identifies
Genes Involved in Karmellae Biogenesis. Jennifer Loertscher,
Emily Cadera, Dangelei Fox, Clint Matson, Jeffrey
Shaver, Christine Tachibana, Robin Wright (manuscript in preparation).
Exposure to 2,3,7,8-Tetrachlorodibenzo-p-dioxin (TCDD) Causes Accelerated
Terminal Differentiation in Fetal Mouse Skin. Loertscher, J. A., T. M. Lin., R.
E. Peterson, B. L. Allen-Hoffmann. (2002) Toxicological Science
Treatment of Normal
Human Keratinocytes With 2,3,7,8-Tetrachlorodibenzo-p-dioxin (TCDD) Causes a
Reduction in Cell Number, But No Increase in Apoptosis. Loertscher J. A., C. M.
Sadek, B. L. Allen-Hoffmann. (2001) Toxicology and Applied Pharmacology
2,3,7,8-Tetrachlorodibenzo-p-dioxin (TCDD) Alters the Differentiation Pattern of
Human Keratinocytes in Organotypic Culture. Loertscher J. A., C. A. Sattler, B.
L. Allen-Hoffmann. (2001) Toxicology and Applied Pharmacology 175,
University of Washington, Biology 401, Seattle,
WA, October 2002, “Ubiquitin is involved in HMG-CoA Reductase-Induced
Endoplasmic Reticulum Biogenesis.”
Yeast Genetics and
Molecular Biology Meeting, Madison, WI, July-August 2002, “A low-tech,
high-throughput screen of the deletion mutant collection efficiently identifies
karmellae biogenesis genes.”
Carroll College, Waukesha, WI, May 2000, “The
Effects of Dioxin on Human and Mouse Skin.”
Poster Presentations at
National Scientific Meetings
Functions of Ubiquitin and Ubiquitin-like Proteins, poster presentation, August
2002, Colorado Springs, CO. Ubiquitin is involved in endoplasmic reticulum
biogenesis in a proteasome-independent manner. Jennifer Loertscher, Christine
Tachibana, Emily Cadera, Mark Parrish, Lynelle Larson, Dangelei Fox, Robin
Toxicology national meeting, poster presentation, March 2001, San Francisco, CA.
The effects of 2,3,7,8-Tetrachlorodibenzo-p-dioxin (TCDD) on keratinocytes in
organotypic cultures follows a structure-activity relationaship. J. A.
Loertscher, C. A. Sattler, B. L. Allen-Hoffmann.
Toxicology national meeting, poster presentation, March 2000, Philadelphia, PA.
2,3,7,8-Tetrachlorodibenzo-p-dioxin (TCDD) causes accelerated differentiation,
but not apoptosis, in keratinocytes in organotypic culture. J. A. Loerstcher, C.
A. Sattler, and B. L. Allen-Hoffmann.
Toxicology national meeting, poster presentation, March 1999, New Orleans, LA.
2,3,7,8-Tetrachlorodibenzo-p-dioxin (TCDD) causes alterations in differentiation
in keratinocytes in organotypic culture. J. A. Loertscher, C. A. R. Ivarie, M.
A. Weitzel, and B. L. Allen-Hoffmann.
Toxicology national meeting, poster presentation, March 1998, Seattle, WA. The
Ah Receptor is not required for suspension-induced apoptosis in keratinocytes.
J. A. Loertscher, M. A. Weitzel, C. M. Sadek, and B. L. Allen-Hoffmann.